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NEW ALBANY, IN (WAVE) - The capital murder trial of an accused serial killer in Southern Indiana is set to begin.

Opening statements will start Monday in Floyd County. William Clyde Gibson, 54, is charged with killing three women over the course of a decade.

Gibson is first on trial for the April 2012 murder of Christine Whitis, 75, of Clarksville and if convicted could face the death penalty. Back in August, it was ruled Gibson is competent to stand trial.

In addition to the murder of Whitis, Gibson is accused of murdering Stephanie Kirk, 35, of Charlestown, Indiana, in 2012, and Karen Hodella, 45, of Port Orange, Florida, in 2002.

Because of pre-trial publicity, prosecutors and defense attorneys completed jury selection last month in Dearborn County near Cincinnati. The jury was picked from a pool of 500 and will be brought to New Albany and sequestered during the trial.

On April 19, 2012, Whitis' body was found inside Gibson's garage on Woodbourne Drive in New Albany. That's when the case against Gibson started unraveling. Whitis was Gibson's mother's long time friend.

Gibson was arrested later that day at the Wal-Mart on Grant Line Road in New Albany. He was originally charged with operating a vehicle while intoxicated and resisting arrest.

Gibson is accused of sexually assaulting Whitis and Prosecutor Keith Henderson says he then dismembered her body by cutting of her breast. Police believe Whitis was strangled.

One week later Kirk was discovered buried in Gibson's backyard.

After his arrest for that murder, police say Gibson also confessed to killing Hodella in 2002. A few days later, he was charged with murdering Kirk who had been missing for more than a month. Her body was discovered buried in Gibson's back yard.

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Accused serial killer trial will begin Monday

You have been hosting reality shows for quite some time, and now you have taken a turn towards the world of movies. If given a chance what would you pick? Acting or anchoring? I dont think I can choose just one, I love both and I want to do both. A lot of people are thinking that now that Ive entered films, I will quit TV. Theyre wrong. I am not going to quit TV. I will definitely be hosting more shows. And I will be doing movies too. I am going to try and create a balance between the both.

Your co-star for your upcoming movie, Elli Avram, is an inmate in the Bigg Boss house. Will it be safe to say that it was an intentional promotional strategy for Mickey Virus? Yes, it definitely was a promotional strategy. Saurabh Varma, the director of the movie, thought it would be a good strategy to promote the film, and the new face of the industry, Elli Avram this way.

What have been some of the most memorable moments from Mickey Virus? I think the entire film has been really memorable because this is my first film. But then Mickey Virus is very close to my heart for a few other reasons too. Firstly, the titular character is named after my real-life nickname, which is Mickey. And secondly, the movies been shot in locations where I grew up, which again made the entire experience very memorable.

While anchoring a show, do you stick to the script or do you improvise on your lines? I manipulate my lines a lot. My writer Siddharth Dey writes brilliant situations for me but he also gives me the liberty to add something if I want to. So, the way I look at it, it is a lot of team effort really. That said, I do end up doing things on the spot which, luckily for me, people like. One of the best things about Jhalak Dikhla Jaa was your chemistry with Kapil Sharma. When are we going to see more of the two of you? Hopefully, very soon. We are dying to work with each other again because, we share great camaraderie and are very good friends in real life. Everybody loved us together. On the sets of Jhalak, Kapil was looked upon as the funny guy and I was called the lady killer on the show. We have a great timing together, so lets see, I am hoping to work with Kapil soon.

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The humour virus

6 hours ago

Each small step that Science takes to discover how viruses infect cells is always very valuable to researchers and society, since it provides relevant information to fight infections.

However, the recent discovery made by the Ikerbasque Professor Nicola Abrescia, at the Center for Cooperative Research CIC bioGUNE, in collaboration with the University of Helsinki, has been more than just a small step. For the first time, a new mechanism used by viruses to infect cells has been described. This mechanism involves the formation of a tube generated with pre-existing lipids and proteins in the structure of the virus. This study, carried out with a virus model system bacteriophage (infects bacteria) PRD1 leads the way forward in the fight against bacterial infections such as E. coli or Salmonella.

The breakthrough is also very important since the virus that has been analysed is representative of other viruses that infect all the kinds of existing cells: archaea, bacteria and eukaryotic. In other words, they can infect animal and plant cells, bacteria and other types of cells.

Virus with lipid vesicle

Unlike bacteria, which are classified as an independent cell domain, viruses are acellular structures that depend on their ability to infect cells and transfer their genetic material in order to ensure their survival and propagation, as they are not able to reproduce on their own. They are basically composed of a genome (DNA or RNA) and a capsid, in other words, a protein structure that envelopes and protects this genetic material. Some have a lipid vesicle enveloping the capsid and, in other cases, the vesicle is surrounded by the capsid.

Research at the CIC bioGUNE, in collaboration with other European centres, has discovered a new viral phenomenon in which viruses with internal lipid vesicles and specifically bacteriophage PRD1, representative of this virus family, injects its genome into the cell to infect it.

This study has earned its publication in the prestigious scientific journal, PLoS Biology, and has been included as one the most outstanding papers in the Synopsis section. The research team observed how the virus develops a proteolipid tail to create a conductive tube, penetrates the bacterial cell and transfers its genome into it in order to replicate.

Research conclusions

One of the main conclusions of the study is that bacteriophage PRD1 uses the proteins of the membrane in its lipid vesicle and the lipids to assemble the tube. Then it drills a hole in the bacteria, which is an essential step to infect it. Researchers also observed that the tube, one of the smallest that has been studied, only allows the passage of one double-stranded DNA molecule.

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Team discovers new form of virus reproduction

Worcestershire owners urged to be on their guard

11:10am Tuesday 15th October 2013 in Ledbury By Gema Bate

A KILLER disease is spreadingamong dogs in Worcestershire.

Owners are being urged notto neglect their pets healthafter four out of 77 strayshave been found to haveparvo, or canine parvovirus,a highly-contagious diseaseamong dogs which can lead toa painful and horrific death.

Symptoms include bloodydiarrhoea, sickness, lethargyand dogs being put off theirfood.

Worcestershire RegulatoryServices dog warden, PipSingleton, said: The factthat four cases have beenidentified means that parvomust be prevalent in the area.

All we can presume is thatbecause of the current economicclimate, dog ownersare not getting their petsproperly vaccinated.

Parvo is a vile, disgustingdisease which, although notcontagious between humans,is highly so among the caninepopulation.

Not vaccinating your petsis not a good money-savingoption, as they will becomevery ill which will mean highvet bills, although parvo, inmost cases, leads to death.

The dogs found to have thedisease were picked up inWorcester, Redditch, Eveshamand Hartlebury andwere aged two weeks, fivemonths, 15 months and two-years-old.

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Killer disease spreading among dogs

Oct. 12, 2013 Development of a vaccine against Epstein-Barr virus (EBV) has taken a step forward with the Canadian discovery of how EBV infection evades detection by the immune system.

EBV causes infectious mononucleosis and cancers such as Hodgkin's lymphoma and nasopharyngeal carcinoma, which is the most common cancer in China, as well as opportunistic cancers in people with weakened immune systems. A member of the herpes virus family that remains in the body for life, the virus infects epithelial cells in the throat and immune cells called B cells.

The researchers discovered that the virus triggers molecular events that turn off key proteins, making infected cells invisible to the natural killer T (NKT) immune cells that seek and destroy EBV-infected cells.

"If you can force these invisible proteins to be expressed, then you can render infected cells visible to NKT cells, and defeat the virus. This could be key to making a vaccine that would provide immunity from ever being infected with EBV," says Dr. Rusung Tan, the study's principal investigator. Dr. Tan is a scientist and director of the Immunity in Health & Disease research group at the Child & Family Research Institute at BC Children's Hospital, and a professor in the Department of Pathology at the University of British Columbia.

The findings were published this week in the print edition of the scientific journal Blood.

For this study, the researchers looked at cells from infected tonsils that had been removed from patients at BC Children's Hospital by Dr. Frederick Kozak. The researchers infected the tonsillar B cells with EBV, and then combined some of these cells with NKT cells. They found that more NKT cells led to fewer EBV-infected cells, while an absence of NKT cells was associated with an increase in EBV-infected cells.

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Immune system discovery could lead to vaccine to prevent mono, some cancers